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Clenbuterol is a substituted phenylaminoethanol that has beta-2 adrenomimetic properties at very low doses. It is used as a bronchodilator in asthma. Although approved for use in some countries, as of fall, 2006, clenbuterol is not an ingredient of any therapeutic drug approved by the U. Food and Drug Administration. Clenbuterol injection improved glucose tolerance by increasing insulin secretion in lean mice. In SKM, clenbuterol stimulated glycogen breakdown. Conclusion: Propranolol, a commonly prescribed β-blocker, reduced weight, increased left ventricular mass and decreased glycogen clearance in skeletal muscle following ERT. Β-Blockers might therefore decrease the efficacy from ERT in patients with Pompe disease. PMID: 11842069 DOI: 10. 2001 Abstract In the present study, we investigated the effects of chronic clenbuterol treatment on insulin-stimulated glucose uptake in the presence of epinephrine in isolated rat skeletal muscle. Clenbuterol (WHO Food Additives Series 38) ADRENOCEPTOR AGONISTS CLENBUTEROL First draft prepared by Dr K. Woodward Veterinary Medicines Directorate Ministry of Agriculture, Fisheries and Food Addlestone, Surrey, United Kingdom 1. 1 Biochemical aspects 2. 1 Absorption, distribution and excretion 2. Clenbuterol-fed rats (2 mg/kg diet for 10 days, providing an average of 0. 04 mg clenbuterol/kg/d) increased body weight (7%), muscle mass (15% to 21%), and muscle protein content (9% to 26%), whereas epididymal fat pad weight and muscle glycogen content were reduced Despite government crackdowns and public health campaigns, the illegal use of this drug continues in the pork industry, putting the health of millions at risk, clenbuterol glycogen.
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